A. Since many patients respond well to immunosuppressive treatment, the recognition of these disorders is of utmost importance. In our report, we present a case of a rare and very severe form of listeriosis-rhombencephalitis in a 61-year-old female with no history of immunosuppression, who, because of history, clinical picture, and laboratory results as well as negative cultures, was at first diagnosed with viral encephalitis. His sodium level (126 mmol/L) was moderately low. Apart from sinusitis no pre-existing medical conditions were known. In our report, we present a case of a rare and very severe form of listeriosis-rhombencephalitis in a 61-year-old female with no history of immunosuppression, who, because of history, clinical picture, and laboratory results as well as negative cultures, was at first diagnosed with viral encephalitis. His sodium level (126 mmol/L) was moderately low.
A good starting point for making therapeutic decisions in the post-TURP or posthysteroscopy setting is calculation of the osmolar gap.6 The osmolar gap is the difference between the actual measured osmolality (as determined by an osmometer) and the calculated osmolality, which is obtained by use of the following formula: 2(serum sodium concentration) + (serum glucose level)/18 + (blood urea nitrogen)/2.4 + (blood ethanol level)/4.6. In our report, we present a case of a rare and very severe form of listeriosis-rhombencephalitis in a 61-year-old female with no history of immunosuppression, who, because of history, clinical picture, and laboratory results as well as negative cultures, was at first diagnosed with viral encephalitis. Two received a saltwater solution within an hour of the seizures, he said, and recovered, although one has permanent double vision. Therefore, we made a presumptive diagnosis of CJD according to the process together with elevated 14-3-3 protein in the CSF,4,5 but without periodic paroxysmal sharp wave complexes (PSWCs) as characteristic EEG findings.6 One week later, we performed a follow-up EEG that showed typical PSWCs (). A 65-year-old Chinese man presented with slowly progressive cognitive decline with psychiatric symptoms. Median follow-up was for 48 months (range = 19–95) with no differences between groups (p = 0.77, Kruskal–Wallis test). Myoclonus appeared in his right upper and lower limbs, and his Parkinsonism worsened.
2). An electroencephalogram was mild to moderately abnormal, showing diffuse slow activity without epileptiform discharges. On arrival, the patient’s temperature was 39.4 °C; she was intubated and sedated. Iizuka T, Sakai F (2008) [Anti-nMDA receptor encephalitis–clinical manifestations and pathophysiology]. She also developed ophthalmoplegia, specifically sixth cranial nerve palsy. Tests for paraneoplastic-associated antibodies in the CSF, such as anti-Ho, anti-Yo, anti-Ri, and anti-CV2, were also negative. It suggests the possible but not essential involvement of hyponatremia in the pathogenesis of M.pneumoniae-induced MERS.
Serum, urine amino acid, and urine organic acid analyses were normal, as was a direct investigation of lymphocytic respiratory chain complex activities. He looked confused, uttered meaningless or incoherent words and phrases and could not respond to questions in an appropriate manner. What is “Cold Diuresis”? The patient died on hospital day 14. The patient was living in an endemic area of tick-borne diseases (tick-borne encephalitis and Lyme disease). CSWS and SIADH share many similar laboratory and clinical findings at initial presentation. She was moved from general ward to intensive care unit, in that hospital, in view of these developments while diarrhea and vomiting resolved.
2. The clinical syndrome associated with GABA(b)-receptor antibodies is a limbic encephalitis (memory dysfunction, behavioural abnormalities and seizures) in which seizures and status epilepticus are especially prominent.7 The patients’ median age is 60 years and men and women are affected equally. The incidence and severity of these neurologic manifestations was significantly lower after treatment with minocycline throughout the course of the disease (A). It is important to screen patients with subacute encephalitis for anti-TPO antibody and VGKCAb, particularly in the presence of seizures. Although SIADH associated with limbic encephalitis is rare, this is not the first reported case in the literature.31, 32, and 33 In this instance, resolution of the hyponatraemia provided diagnostic clarity, as the persistence of symptoms indicated an underlying cerebral pathology. These unusual disorders offer the opportunity of developing an insight into the autoimmune disorders. The presence of large radiographic lesions did not predict a poor outcome, but either high cerebrospinal fluid white-cell counts or severe hyponatremia did.
The dengue was diagnosed on the basis of the positive serum IgM antibody to dengue virus by the IgM antibody capture enzyme-linked immunosorbent assay (MAC ELISA) method with NOVATEC Kit. was convincingly substantiated . Seizures may confound interpretation of the MRI, as they can produce T2 fluid attenuation inversion recovery (FLAIR) hyperintensities, contrast enhancement, and even diffusion restriction.28 Some studies have reported brain positron emission tomography (PET) findings that may aid in making the diagnosis of autoimmune encephalitis, but further analyses remain to be done with larger numbers of patients.29 The EEG can have obvious abnormalities but sometimes findings may be subtle,30 so the interpreting physician should be made aware of the clinical picture and the suspicion of autoimmune encephalitis.